19 results on '"Gumperz J"'
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2. The Communicative Bases of Social Inequality
3. La grille de codage
4. Friday, August 31, 1962 (at Harvard)
5. The Kaposi's Sarcoma Associated Herpesvirus Modulators of Immune Recognition (MIR) Proteins Influence Innate Immunity by Downregulating CD1d
6. Natural inactivation of a common HLA allele (A*2402) has occurred on at least three separate occasions.
7. Conserved and variable residues within the Bw4 motif of HLA-B make separable contributions to recognition by the NKB1 killer cell-inhibitory receptor.
8. Polymorphism in the alpha 1 helix of the HLA-B heavy chain can have an overriding influence on peptide-binding specificity.
9. Peptides bound endogenously by HLA-Cw*0304 expressed in LCL 721. 221 cells include a peptide derived from HLA-E
10. Specificity of two anti‐class IHLA monoclonal antibodies that block class I recognition by the NKB1 killer cell inhibitory receptor
11. The inter-locus recombinant HLA-B*4601 has high selectivity in peptide binding and functions characteristic of HLA-C.
12. Unusual uniformity of the N-linked oligosaccharides of HLA-A, -B, and -C glycoproteins.
13. Heterogeneous phenotypes of expression of the NKB1 natural killer cell class I receptor among individuals of different human histocompatibility leukocyte antigens types appear genetically regulated, but not linked to major histocompatibililty complex haplotype.
14. Expression of an unusual Bw4 epitope by a subtype of HLA‐B8 [B*0802]
15. Low HLA-C expression at cell surfaces correlates with increased turnover of heavy chain mRNA.
16. The NKB1 and HP-3E4 NK cells receptors are structurally distinct glycoproteins and independently recognize polymorphic HLA-B and HLA-C molecules.
17. The Bw4 public epitope of HLA-B molecules confers reactivity with natural killer cell clones that express NKB1, a putative HLA receptor.
18. NKB1: a natural killer cell receptor involved in the recognition of polymorphic HLA-B molecules.
19. Specificity of HLA class I antigen recognition by human NK clones: evidence for clonal heterogeneity, protection by self and non-self alleles, and influence of the target cell type.
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