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1. Correction: Synaptic and memory dysfunction induced by tau oligomers is rescued by up-regulation of the nitric oxide cascade

8. List of Contributors

12. A novel arousal-based individual screening reveals susceptibility and resilience to PTSD-like phenotypes in mice

16. Synaptic and memory dysfunction induced by tau oligomers is rescued by up-regulation of the nitric oxide cascade

17. Neuromodulatory Action of Picomolar Extracellular Aβ42 Oligomers on Presynaptic and Postsynaptic Mechanisms Underlying Synaptic Function and Memory

18. Role of Amyloid-β and Tau Proteins in Alzheimer’s Disease: Confuting the Amyloid Cascade

19. Dopaminergic-GABAergic interplay and alcohol binge drinking

21. Activation of Serotonin 5-HT7 Receptors Modulates Hippocampal Synaptic Plasticity by Stimulation of Adenylate Cyclases and Rescues Learning and Behavior in a Mouse Model of Fragile X Syndrome

23. Role of Amyloid-β and Tau Proteins in Alzheimer’s Disease: Confuting the Amyloid Cascade

25. LTP and memory impairment caused by extracellular Aβ and Tau oligomers is APP-dependent

26. Author response: LTP and memory impairment caused by extracellular Aβ and Tau oligomers is APP-dependent

27. Amyloid-β Peptide Is Needed for cGMP-Induced Long-Term Potentiation and Memory

29. CL316,243, a β3-adrenergic receptor agonist, induces muscle hypertrophy and increased strength

30. Time-dependent reversal of synaptic plasticity induced by physiological concentrations of oligomeric Aβ42: an early index of Alzheimer’s disease

32. The antineoplastic drug flavopiridol reverses memory impairment induced by Amyloid-ß 1-42 oligomers in mice

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