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13. Data from A Three-Gene Expression Signature Model for Risk Stratification of Patients with Neuroblastoma

14. Supplementary Figure from Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

15. Supplementary Table 5 from A Three-Gene Expression Signature Model for Risk Stratification of Patients with Neuroblastoma

16. Supplementary Table from Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

17. Supplementary Table from Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

18. Supplementary Table 5 from A Three-Gene Expression Signature Model for Risk Stratification of Patients with Neuroblastoma

19. Supplementary Table 1 from A Three-Gene Expression Signature Model for Risk Stratification of Patients with Neuroblastoma

20. Supplementary Table from Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

21. Supplementary Table from Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

22. Supplementary Figures 1-11, Tables 2-4 from A Three-Gene Expression Signature Model for Risk Stratification of Patients with Neuroblastoma

23. Supplementary Figure from Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

24. Supplementary Table from Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

25. Supplementary Figures 1-11, Tables 2-4 from A Three-Gene Expression Signature Model for Risk Stratification of Patients with Neuroblastoma

26. Supplementary Table 1 from A Three-Gene Expression Signature Model for Risk Stratification of Patients with Neuroblastoma

27. Supplementary Table from Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

28. Data from Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

29. Data from A Three-Gene Expression Signature Model for Risk Stratification of Patients with Neuroblastoma

32. Author Correction: PR-LncRNA signature regulates glioma cell activity through expression of SOX factors

35. Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

36. Abstract #10 PR-LncRNA Signature May Regulates Glioma Cell Activity Through Expression of SOX Factors

45. SOX2 expression diminishes with ageing in several tissues in mice and humans

46. SOX3 can promote the malignant behavior of glioblastoma cells

47. PR-LncRNA signature regulates glioma cell activity through expression of SOX factors

49. Coupling of Magnetothermal and Mechanical Superconducting Magnet Models by Means of Mesh-Based Interpolation

50. High expression of MKP1/DUSP1 counteracts glioma stem cell activity and mediates HDAC inhibitor response

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