38 results on '"Cornelius, Ryan J."'
Search Results
2. Enriched Single-Nucleus RNA-Sequencing Reveals Unique Attributes of Distal Convoluted Tubule Cells
3. Enriched Single-Nucleus RNA-Sequencing reveals unique attributes of distal convoluted tubule cells
4. BK Channels in Epithelia
5. Cullin 3 and Blood Pressure Regulation: Insights From Familial Hyperkalemic Hypertension
6. Renal effects of cullin 3 mutations causing familial hyperkalemic hypertension
7. Cullin 3 mutant causing familial hyperkalemic hypertension lacks normal activity in the kidney
8. COP9 signalosome deletion promotes renal injury and distal convoluted tubule remodeling
9. DCT‐specific COP9 Signalosome Deletion Activates the WNK4‐NCC Pathway and Mimics Familial Hyperkalemic Hypertension
10. Combined Kelch-like 3 and Cullin 3 Degradation is a Central Mechanism in Familial Hyperkalemic Hypertension in Mice
11. BK Channels in Epithelia
12. A novel distal convoluted tubule-specific Cre-recombinase driven by the NaCl cotransporter gene
13. A Novel Distal Convoluted Tubule‐Specific Tamoxifen‐Inducible Cre‐Recombinase Driven by the NaCl Cotransporter Gene
14. Hypertension-causing cullin 3 mutations disrupt COP9 signalosome binding
15. WNK bodies cluster WNK4 and SPAK/OSR1 to promote NCC activation in hypokalemia
16. Cullin-Ring ubiquitin ligases in kidney health and disease
17. Long‐term Disruption of the COP9 Signalosome Decreases NCC Abundance Due to Remodeling of the DCT
18. Renal COP9 Signalosome Deficiency Alters CUL3-KLHL3-WNK Signaling Pathway
19. With no lysine kinase 4 modulates sodium potassium 2 chloride cotransporter activity in vivo
20. Dual gain and loss of cullin 3 function mediates familial hyperkalemic hypertension
21. Kidney‐Specific COP9 Signalosome (CSN) Deletion Mimics FHHt Effects on WNK/NCC Signaling
22. Dual Gain and Loss of Cullin 3 Function Mediates Familial Hyperkalemic Hypertension
23. Neddylation‐Deficient K712R CUL3 Δ403‐459 Double Mutant Attenuates Proteasome‐Dependent KLHL3 Degradation
24. A Micropuncture Study of the Net K Secretion in Thick Ascending Limb of Mice on a Low Na High K Diet
25. Maintaining K+ balance on the low-Na+, high-K+ diet
26. Deficient acid handling with distal RTA in the NBCe2 knockout mouse
27. Increased Epithelial Sodium Channel Activity Contributes to Hypertension Caused by Na + -HCO 3 − Cotransporter Electrogenic 2 Deficiency
28. Low Na, High K Diet and the Role of Aldosterone in BK-Mediated K Excretion
29. Relation between BK-α/β4-mediated potassium secretion and ENaC-mediated sodium reabsorption
30. Interacting influence of diuretics and diet on BK channel-regulated K homeostasis
31. Regulation of BK-α expression in the distal nephron by aldosterone and urine pH
32. Aldosterone (aldo) and epithelial Na channel (ENaC)‐ dependent K secretion by BK‐α/β4 in the distal nephron (DN)
33. Bicarbonate promotes BK-α/β4-mediated K excretion in the renal distal nephron
34. Bicarbonate transport drives Na‐independent K secretion (NIKS) via BK‐α/β4 in β‐intercalated cells (IC‐β)
35. The trans‐tubular K gradients (TTKG) reveal long‐term adaptation in mice to Na deficient, high K diets
36. Role of Aquaporin2 (Aqp2) in the increased urinary flow of K‐adapted mice
37. Coupled ATP and potassium efflux from intercalated cells
38. Mediation of Na‐independent K secretion by intercalated cell BK‐α/β4
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.