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1. Glomerular Elasticity and Gene Expression Patterns Define Two Phases of Alport Nephropathy

3. Viral associations with kidney disease diagnosis and altered kidney metatranscriptome by kidney function

11. Podocyte density is reduced in kidney allografts with high‐risk APOL1 genotypes at transplantation

20. Similar Biophysical Abnormalities in Glomeruli and Podocytes from Two Distinct Models

22. ApoL1 Overexpression Drives Variant-Independent Cytotoxicity

30. Biochemical and Cellular Determinants of Renal Glomerular Elasticity

34. Nephropathy in HIV-Transgenic Mice

35. Executive Summary: Clinical Practice Guideline for the Management of Chronic Kidney Disease in Patients Infected With HIV: 2014 Update by the HIV Medicine Association of the Infectious Diseases Society of America

36. Clinical Practice Guideline for the Management of Chronic Kidney Disease in Patients Infected With HIV: 2014 Update by the HIV Medicine Association of the Infectious Diseases Society of America

37. Plasma Apolipoprotein L1 Levels Do Not Correlate with CKD

42. Familial Focal Segmental Glomerulosclerosis (FSGS)-linked α-Actinin 4 (ACTN4) Protein Mutants Lose Ability to Activate Transcription by Nuclear Hormone Receptors

46. Identification of the Nephropathy-Susceptibility Locus HIVAN4

47. Biophysical properties of normal and diseased renal glomeruli

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