17 results on '"Boninsegna, S"'
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2. 524 RESPONSE-GUIDED PEGINTERFERON ALFA-2A THERAPY BASED ON HBSAG LEVELS AT WEEK 12 AND WEEK 24 OR 48 IMPROVES RESPONSE RATES IN HBEAG-NEGATIVE, GENOTYPE D PATIENTS
3. Viral kinetics during the first weeks of pegylated interferon and ribavirin treatment can identify patients at risk of relapse after its discontinuation: new strategies for such patients?
4. 132 ALLOGRAFT EXPRESSION OF INNATE IMMUNITY GENES INFLUENCES LIVER FIBROSIS PROGRESSION AND ACUTE CELLULAR REJECTION IN LIVER TRANSPLANTED PATIENTS
5. 98 EXTENDED (2 YEARS) TREATMENT WITH PEGINTERFERON ALFA-2A [40KD] IMPROVES SUSTAINED RESPONSE RATES IN GENOTYPE D PATIENTS WITH HBEAG NEGATIVE CHRONIC HEPATITIS B
6. 682 INNATE IMMUNE SYSTEM RESPONSES DIFFER DURING RECENT AND CHRONIC HEPATITIS C VIRUS INFECTION
7. 28 ALLOGRAFT EXPRESSION OF INNATE IMMUNITY GENES INFLUENCES LIVER FIBROSIS PROGRESSION AND ACUTE CELLULAR REJECTION IN LIVER TRANSPLANTED PATIENTS
8. Genotypes 2 and 3 recurrent hepatitis C after liver transplantation: Excellent results with suboptimal doses of antiviral therapy
9. Histological HCV recurrence after liver transplantation (LT): The impact of antiviral therapy on fibrosis progression
10. HCV Histological Recurrence and Survival Following Liver Transplantation in Patients With and Without Hepatocellular Carcinoma
11. PA.191 COMBINED ANTIVIRAL THERAPY(PEG-IFN +RIBAVIRINE) IN RECURRENT HEPATITIS C AFTER LIVER TRANSPLANTATION. AN ITALIAN EXPERIENCE OF TWO TRANSPLANT CENTRES
12. Antiviral Therapy for Hepatitis C Virus Recurrence Following Liver Transplantation: Long-Term Results From a Single Center Experience
13. Predictive factors of HCV recurrence after liver transplantation (OLTX)
14. Encapsulation of individual pancreatic islets by sol–gel SiO2
15. SiO2 Entrapment of animal cells. Part 2: Protein diffusion through collagen membranes coated with sol–gel SiO2
16. Rapid detection of a protein C gene mutation present in the asymptomatic and not in the thrombosis‐prone lineage
17. A recurrent missense mutation (Arg → Gln) and a partial deletion in factor VIII gene causing severe haemophilia A
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