50 results on '"Awad, Milena"'
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2. Clostridium septicum α-toxin activates the NLRP3 inflammasome by engaging GPI-anchored proteins
3. A Highly Specific Holin-Mediated Mechanism Facilitates the Secretion of Lethal Toxin TcsL in Paeniclostridium sordellii
4. PGFinder, a novel analysis pipeline for the consistent, reproducible, and high-resolution structural analysis of bacterial peptidoglycans
5. Author response: PGFinder, a novel analysis pipeline for the consistent, reproducible, and high-resolution structural analysis of bacterial peptidoglycans
6. PGfinder, a novel analysis pipeline for the consistent, reproducible and high-resolution structural analysis of bacterial peptidoglycans
7. Chromosome Segregation and Peptidoglycan Remodeling Are Coordinated at a Highly Stabilized Septal Pore to Maintain Bacterial Spore Development
8. A dynamic, ring-forming MucB / RseB-like protein influences spore shape in Bacillus subtilis
9. Human Plasminogen Exacerbates Clostridioides difficile Enteric Disease and Alters the Spore Surface
10. Paeniclostridium (Clostridium) sordellii–associated enterocolitis in 7 horses
11. Tranexamic Acid Influences the Immune Response, but not Bacterial Clearance in a Model of Post-Traumatic Brain Injury Pneumonia
12. Cephamycins inhibit pathogen sporulation and effectively treat recurrent Clostridioides difficile infection
13. Paeniclostridium sordellii and Clostridioides difficile encode similar and clinically relevant tetracycline resistance loci in diverse genomic locations
14. pCP13, a representative of a new family of conjugative toxin plasmids in Clostridium perfringens
15. Lectin Activity of the TcdA and TcdB Toxins of Clostridium difficile
16. Clostridium sordellii outer spore proteins maintain spore structural integrity and promote bacterial clearance from the gastrointestinal tract
17. Clostridium sordellii Pathogenicity Locus Plasmid pCS1-1 Encodes a Novel Clostridial Conjugation Locus
18. Structural Characterization of Clostridium sordellii Spores of Diverse Human, Animal, and Environmental Origin and Comparison to Clostridium difficile Spores
19. Functional analysis of an feoB mutant in Clostridium perfringens strain 13
20. The NEAT Domain-Containing Proteins of Clostridium perfringens Bind Heme
21. The Sialidase NanS Enhances Non-TcsL Mediated Cytotoxicity of Clostridium sordellii
22. Disruption of the Gut Microbiome: Clostridium difficile Infection and the Threat of Antibiotic Resistance
23. Necrotic Enteritis in Chickens Associated withClostridium sordellii
24. Clostridium sordellii genome analysis reveals plasmid localized toxin genes encoded within pathogenicity loci
25. Antibiotic resistance, virulence factors and genetics of Clostridium sordellii
26. The Pore-Forming α-Toxin from Clostridium septicum Activates the MAPK Pathway in a Ras-c-Raf-Dependent and Independent Manner
27. Comparing the identification of Clostridium spp. by two Matrix-Assisted Laser Desorption Ionization-Time of Flight (MALDI-TOF) mass spectrometry platforms to 16S rRNA PCR sequencing as a reference standard: A detailed analysis of age of culture and sample preparation
28. Expression of the large clostridial toxins is controlled by conserved regulatory mechanisms
29. Clostridium difficilevirulence factors: Insights into an anaerobic spore-forming pathogen
30. Utility of the Clostridial Site-Specific Recombinase TnpX To Clone Toxic-Product-Encoding Genes and Selectively Remove Genomic DNA Fragments
31. Towards an understanding of the role of Clostridium perfringens toxins in human and animal disease
32. Opioid Analgesics Stop the Development of Clostridial Gas Gangrene
33. The Cysteine Protease α-Clostripain is Not Essential for the Pathogenesis of Clostridium perfringens-Mediated Myonecrosis
34. TcsL Is an Essential Virulence Factor in Clostridium sordellii ATCC 9714
35. TcdB or not TcdB: a tale of two Clostridium difficile toxins
36. Novel Use of Tryptose Sulfite Cycloserine Egg Yolk Agar for Isolation of Clostridium perfringens during an Outbreak of Necrotizing Enterocolitis in a Neonatal Unit
37. The NanI and NanJ Sialidases of Clostridium perfringens Are Not Essential for Virulence
38. Functional Analysis of the VirSR Phosphorelay from Clostridium perfringens
39. Molecular and Cellular Basis of Microvascular Perfusion Deficits Induced by Clostridium perfringens and Clostridium septicum
40. Alpha-Toxin of Clostridium perfringens Is Not an Essential Virulence Factor in Necrotic Enteritis in Chickens
41. Skewed genomic variability in strains of the toxigenic bacterial pathogen, Clostridium perfringens
42. The α-toxin ofClostridium septicumis essential for virulence
43. The FxRxHrS Motif: A Conserved Region Essential for DNA Binding of the VirR Response Regulator from Clostridium perfringens
44. Perfringolysin O Expression in Clostridium perfringens Is Independent of the Upstream pfoR Gene
45. Synergistic Effects of Alpha-Toxin and Perfringolysin O in Clostridium perfringens -Mediated Gas Gangrene
46. Use of Genetically Manipulated Strains of Clostridium perfringens Reveals that Both Alpha-Toxin and Theta-Toxin Are Required for Vascular Leukostasis To Occur in Experimental Gas Gangrene
47. Clostridial Gas Gangrene: Evidence That α and θ Toxins Differentially Modulate the Immune Response and Induce Acute Tissue Necrosis
48. Isolation of α-toxin, θ-toxin and κ-toxin mutants ofClostridium perfringensby Tn916mutagenesis
49. The Level of Expression of α-toxin by Different Strains ofClostridium perfringensis Dependent on Differences in Promoter Structure and Genetic Background
50. Identification and molecular analysis of a locus that regulates extracellular toxin production in Clostridium perfringens
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