Search

Equations based on surface renewal (SR) analysis to estimate the sensible heat flux (H) require as input the mean ramp amplitude and period observed in the ramp‐like pattern of the air temperature measured at high frequency. A SR‐based method to estimate sensible heat flux (HSR‐LST) requiring only low‐frequency measurements of the air temperature, horizontal mean wind speed, and land‐surface temperature as input was derived and tested under unstable conditions over a heterogeneous canopy (olive grove). HSR‐LSTassumes that the mean ramp amplitude can be inferred from the difference between land‐surface temperature and mean air temperature through a linear relationship and that the ramp frequency is related to a wind shear scale characteristic of the canopy flow. The land‐surface temperature was retrieved by integrating in situ sensing measures of thermal infrared energy emitted by the surface. The performance of HSR‐LSTwas analyzed against flux tower measurements collected at two heights (close to and well above the canopy top). Crucial parameters involved in HSR‐LST, which define the above mentioned linear relationship, were explained using the canopy height and the land surface temperature observed at sunrise and sunset. Although the olive grove can behave as either an isothermal or anisothermal surface, HSR‐LSTperformed close to Hmeasured using the eddy covariance and the Bowen ratio energy balance methods. Root mean square differences between HSR‐LSTand measured Hwere of about 55 W m−2. Thus, by using multitemporal thermal acquisitions, HSR‐LSTappears to bypass inconsistency between land surface temperature and the mean aerodynamic temperature. The one‐source bulk transfer formulation for estimating Hperformed reliable after calibration against the eddy covariance method. After calibration, the latter performed similar to the proposed SR‐LST method. A method to estimate the sensible heat flux, H, based on surface renewal, SR, is proposed, HSRHSR does not requires high‐frequency measurements as inputBecause HSR involves the land surface temperature can be used in remote sensing

We are proposing a new method for estimating soil surface water content from thermal inertia distributions retrieved from visible–near infrared (VIS-NIR) and thermal infrared (TIR) images. A drying experiment was conducted on three fine-textured soils while acquiring multispectral VIS-NIR and TIR images. Simultaneous measurements of soil water content and thermal inertia were conducted by the thermogravimetric method and the heat pulse technique, respectively. Direct measurements were used to test the thermal inertia approach proposed by Murray and Verhoef that requires only knowledge of soil porosity and can be easily inverted to derive soil water content from thermal inertia. For the three considered soils, the performance of the Murray and Verhoef model was practically equal to that of the traditional approach based on the direct estimation of thermal conductivity and heat capacity, which requires more detailed information about soil properties. With the aim of simplifying the estimation of thermal inertia from remotely sensed images, a modified Kersten function was proposed in which the normalized thermal inertia is substituted by the normalized apparent thermal inertia. Comparison between the two modified Kersten functions was satisfactory. The proposed approach allowed predictions of the surface soil moisture from apparent thermal inertia distributions with an acceptable level of accuracy for practical purposes (0.028 = RMSE = 0.043 m3m-3) and therefore it can be considered a simple and effective tool for estimating the spatial and temporal distribution of surface soil moisture from VIS-NIR and TIR remotely sensed data.

The objective of this study was to investigate the long-term effects of anti-retroviral protease inhibitors (PIs) on 2-deoxy-d -glucose (2-DG) transport in L6 cells in vitro. Exposure of L6 cells to saquinavir, ritonavir, indinavir and amprenavir resulted in significant increases in 2-DG transport using PI concentrations of 1-10 microM with continual exposure to PI. After removal of the PI for up to 48 h, 2-DG transport increases did not change and remained at pre-reversal levels. These changes in 2-DG transport were not related to stress-induced sugar transport or to apoptosis. The examination of glucose transporter (GLUT) 1, 3 or 4 translocation with subcellular fractionation indicated that insulin (i.e. 67 nM) could induce the translocation of all the GLUTs to the plasma membrane. Also, ritonavir (10 microM), which leads to a 2-fold increase in 2-DG transport, demonstrated increased GLUT (i.e. 1, 3 or 4) presence in the plasma membrane fraction, in the presence or absence of insulin. This increased 2-DG transport involved transporter presence in plasma membrane preparations and did not affect the ability of insulin to stimulate 2-DG transport with continual PI exposure. The mechanism(s) involved indicates ready reversibility of PI effects on transporters. The mechanism(s) why reversibility of PI-induced 2-DG transport was similar plus or minus PI was not apparent.