39 results on '"Stemmer-Rachamimov, Anat"'
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2. Gene replacement therapy in a schwannoma mouse model of neurofibromatosis type 2
3. Updated diagnostic criteria and nomenclature for neurofibromatosis type 2 and schwannomatosis: An international consensus recommendation
4. Revised diagnostic criteria for neurofibromatosis type 1 and Legius syndrome: an international consensus recommendation
5. Long-Term Therapeutic Efficacy of Intravenous AAV-Mediated Hamartin Replacement in Mouse Model of Tuberous Sclerosis Type 1
6. An Integrative Model of Cellular States, Plasticity, and Genetics for Glioblastoma
7. Defining T Cell States Associated with Response to Checkpoint Immunotherapy in Melanoma
8. Programming of Schwann Cells by Lats1/2-TAZ/YAP Signaling Drives Malignant Peripheral Nerve Sheath Tumorigenesis
9. Anti-VEGF treatment improves neurological function in tumors of the nervous system
10. Gene replacement therapy in a schwannoma mouse model of neurofibromatosis type 2
11. Olig2-Dependent Reciprocal Shift in PDGF and EGF Receptor Signaling Regulates Tumor Phenotype and Mitotic Growth in Malignant Glioma
12. Insertional Mutagenesis Identifies a STAT3/Arid1b/β-catenin Pathway Driving Neurofibroma Initiation
13. Survival benefit and phenotypic improvement by hamartin gene therapy in a tuberous sclerosis mouse brain model
14. A Call for Discovery and Therapeutic Development for Cutaneous Neurofibromas
15. Nf1 Loss and Ras Hyperactivation in Oligodendrocytes Induce NOS-Driven Defects in Myelin and Vasculature
16. Bevacizumab With Angiostatin-armed oHSV Increases Antiangiogenesis and Decreases Bevacizumab-induced Invasion in U87 Glioma
17. Vascular endothelial growth factor (VEGF) isoform regulation of early forebrain development
18. A Tight Junction-Associated Merlin-Angiomotin Complex Mediates Merlin's Regulation of Mitogenic Signaling and Tumor Suppressive Functions
19. Proteasomal and Genetic Inactivation of the NF1 Tumor Suppressor in Gliomagenesis
20. Tissue-Specific Ablation of Prkar1a Causes Schwannomas by Suppressing Neurofibromatosis Protein Production
21. Preventing Growth of Brain Tumors by Creating a Zone of Resistance
22. Plexiform and Dermal Neurofibromas and Pigmentation Are Caused by Nf1 Loss in Desert Hedgehog-Expressing Cells
23. Modeling NF2 with human arachnoidal and meningioma cell culture systems: NF2 silencing reflects the benign character of tumor growth
24. Inactivation patterns of NF2 and DAL-1/4.1B (EPB41L3) in sporadic meningioma
25. Histopathologic evaluation of atypical neurofibromatous tumors and their transformation into malignant peripheral nerve sheath tumor in patients with neurofibromatosis 1—a consensus overview
26. NHE-RF, a Merlin-Interacting Protein, Is Primarily Expressed in Luminal Epithelia, Proliferative Endometrium, and Estrogen Receptor-Positive Breast Carcinomas
27. Survey of Somatic Mutations in Tuberous Sclerosis Complex (TSC) Hamartomas Suggests Different Genetic Mechanisms for Pathogenesis of TSC Lesions
28. Malignant Transformation of Neurofibromas in Neurofibromatosis 1 Is Associated with CDKN2A/p16 Inactivation
29. Remote acute demyelination after focal proton radiation therapy for optic nerve meningioma
30. Turner syndrome and meningioma: Support for a possible increased risk of neoplasia in Turner syndrome
31. Long-term therapeutic efficacy of intravenous AAV-mediated hamartin replacement in mouse model of tuberous sclerosis type 1
32. Defining T Cell States Associated with Response to Checkpoint Immunotherapy in Melanoma
33. A novel imaging-compatible sciatic nerve schwannoma model
34. Sporadic NF2 Mosaic: Multiple spinal schwannomas presenting with severe, intractable pain following pregnancy
35. Ependymoma of the sella turcica: a variant of pituicytoma
36. Sellar abnormalities in female first-degree relatives
37. 196. AAV-Mediated Gene Replacement Therapy in Mouse Model of Tuberous Sclerosis
38. Conditional Inactivation of SMARCB1 in P0 Permissive Mouse Cells Generates Rhabdoid Tumors in the Peripheral Nervous System
39. The von Hippel-Lindau Tumor Suppressor Protein Is Required for Proper Assembly of an Extracellular Fibronectin Matrix
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