23 results on '"Carretero, Oscar A."'
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2. Role of macula densa adenosine triphosphate (ATP) in tubuloglomerular feedback
3. Superoxide enhances tubuloglomerular feedback by constricting the afferent arteriole
4. Role of angiotensin II type 2 receptors and kinins in the cardioprotective effect of angiotensin II type 1 receptor antagonists in rats with heart failure
5. Dual inhibition of ACE and NEP provides greater cardioprotection in mice with heart failure
6. Role of a selective aldosterone blocker in mice with chronic heart failure
7. Increased intracellular Ca++ in the macula densa regulates tubuloglomerular feedback
8. Glomerular autacoids stimulated by bradykinin regulate efferent arteriole tone
9. Long-term effects of selective and nonselective endothelin receptor antagonists in mice with heart failure
10. Mechanism involved in bradykinin-induced efferent arteriole dilation
11. Role of mesangial cells and gap junctions in tubuloglomerular feedback
12. Possible role of adenosine in macula densa control of glomerular hemodynamics
13. Nitric oxide synthase gene knockout mice do not become hypertensive during pregnancy
14. Efferent arteriole tubuloglomerular feedback in the renal nephron
15. Role of macula densa nitric oxide and cGMP in the regulation of tubuloglomerular feedback
16. Angiotensin II action in isolated microperfused rabbit afferent arterioles is modulated by flow
17. Glomerular prostaglandins modulate vascular reactivity of the downstream efferent arterioles
18. Synthesis of renin by tubulocystic epithelium in autosomal-dominant polycystic kidney disease
19. Characterization of a mouse cortical collecting duct cell line
20. Effects of propranolol on the development of renovascular hypertension in the rat
21. Plasma renin and blood pressure after sympathetic stimulation in normotensive and hypertensive patients
22. Changes in sarcoplasmic reticulum Ca 2+-ATPase and phospholamban during the development of left ventricular hypertrophy in rats
23. Effects of pre-existing left ventricular hypertrophy on ventricular dysfunction and remodeling following myocardial infarction in rats
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