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Your search keyword '"Ventura-Clapier R"' showing total 4 results
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4 results on '"Ventura-Clapier R"'

1. Muscle creatine kinase-deficient mice: II cardiac and skeletal muscles exhibit tissue-specific adaption of the mitochondrial function

Author

Veksler, V.I., Kuznetsov, A.V., Anflous, K., Mateo, P., Deursen, J.M.A. van, Wieringa, B., and Ventura-Clapier, R.

Subjects
Targeted insertional mutagenesis in chromosomal DNA of cell lines and the germ line of mice as means to study gene function, Gerichte mutagenese in chromosomaal DNA van cellijnen en de kiembaan van muizen als middel voor bestudering van genfunctie
Abstract

Contains fulltext : 174479.pdf (Publisher’s version ) (Open Access)

Published
1995

2. Muscle creatine kinase-deficient mice

Author

Ventura-Clapier, R., Kuznetsov, A.V., Albis, A. d', Deursen, J.M.A. van, Wieringa, B., and Veksler, V.I.

Subjects
GeneralLiterature_REFERENCE(e.g.,dictionaries,encyclopedias,glossaries)
Abstract

Contains fulltext : 20682___.PDF (Publisher’s version ) (Open Access)

Published
1995

4. [Contractile properties and creatine kinase activity of myofilaments after ischemia and reperfusion of the rat heart]

Author

Ventura-Clapier R, Vladimir Veksler, Gv, Elizarova, Mekhfi H, and El, Levitskaia

Subjects
Perfusion, Actin Cytoskeleton, Coronary Circulation, Myocardium, Animals, Coronary Disease, Rats, Inbred Strains, In Vitro Techniques, Creatine Kinase, Myocardial Contraction, Cytoskeleton, Rats
Abstract

The state of myofibril creatinkinase and contractile properties of chemically skinned myocardial fibers of rat after 70-90 min ischemia and 15-30 min reperfusion was studied. In spite of sharp fall in the total creatinkinase activity in the tissue, the enzyme activity in myofibrils does not change greatly. Ischemia does not change the functional abilities of myofibril creatinkinase as well as characteristics of Ca-activated contraction of skinned fibers. The results show that irreversible loss of myocardial contractile activity after prolonged ischemia and reperfusion is not connected with violation of myofilament characteristics or deterioration of functional association between myofibril creatinkinase and ATPase.

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