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Supplemental Material, CTO_meta-analysis_R2_Supplemental_appendix for Percutaneous Coronary Intervention With Drug-Eluting Stent Versus Optimal Medical Therapy for Chronic Total Occlusion: Systematic Review and Meta-Analysis by Mohamed Abo-Aly, Naoki Misumida, Neil Backer, Khaled ElKholey, Sun Moon Kim, Gbolahan O. Ogunbayo, Ahmed Abdel-Latif and Khaled M. Ziada in Angiology

Supplemental_Table_1 for Clinical Outcome of Takotsubo Cardiomyopathy Diagnosed With or Without Coronary Angiography by Naoki Misumida, Gbolahan O. Ogunbayo, Sun Moon Kim, Ahmed Abdel-Latif, Khaled M. Ziada, and Vincent L. Sorrell in Angiology

Supplemental_Table_1 for Higher Risk of Bleeding in Asians Presenting With ST-Segment Elevation Myocardial Infarction: Analysis of the National Inpatient Sample Database by Naoki Misumida, Gbolahan O. Ogunbayo, Sun Moon Kim, Odunayo Olorunfemi, Ayman Elbadawi, Richard J. Charnigo, Ahmed Abdel-Latif, and Khaled M. Ziada in Angiology

Previous studies have suggested that diuretic therapy for heart failure may lead to thiamine deficiency due to the increased urinary thiamine excretion. Herein, we present the case of a 61-year-old man with shoshin beriberi, a fulminant form of wet beriberi, induced by long-term diuretic therapy. The patient had a history of heart failure with preserved ejection fraction and was receiving furosemide and trichlormethiazide therapy. He presented with worsening exertional dyspnea and was admitted for heart failure exacerbation. His condition failed to improve even after intensive treatment. A hemodynamic evaluation with the Swan-Ganz catheter revealed high-output heart failure with low peripheral vascular resistance. Thiamine was administered for suspected shoshin beriberi; his hemodynamic status improved dramatically within the next six hours. The serum thiamine level was below the normal range; the patient was therefore diagnosed with shoshin beriberi. The common causes of thiamine deficiency were not identified. Long-term diuretic therapy with furosemide and thiazide was thought to have played a major role in the development of thiamine deficiency. This case illustrates the importance of considering wet beriberi as a possible cause of heart failure exacerbation in patients taking diuretics, even when the common thiamine deficiency causes are not identified with history-taking.