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5. (+)‐Catechin Attenuates Multiple Atherosclerosis‐Associated Processes In Vitro, Modulates Disease‐Associated Risk Factors in C57BL/6J Mice and Reduces Atherogenesis in LDL Receptor Deficient Mice by Inhibiting Inflammation and Increasing Markers of Plaque Stability

6. Molecular MRD Assessment Is Strongly Prognostic in Patients with NPM1 Mutated AML Receiving Venetoclax Based Non-Intensive Therapy

7. Venetoclax-Based Non-Intensive Combinations Successfully Salvage Molecular Relapse of Acute Myeloid Leukemia and Are an Important Bridge to Cellular Therapy in Relapsed/Refractory Disease - Real-World Data from a UK-Wide Programme

8. A multiplex implantable microdevice assay identifies synergistic combinations of cancer immunotherapies and conventional drugs

9. Supplementary Table 4 from The Transcription Factor ZNF217 Is a Prognostic Biomarker and Therapeutic Target during Breast Cancer Progression

10. Data from Exon-Level Microarray Analyses Identify Alternative Splicing Programs in Breast Cancer

11. Supplementary Movie from The Transcription Factor ZNF217 Is a Prognostic Biomarker and Therapeutic Target during Breast Cancer Progression

12. Supplementary Methods, Figures 1-4, Movie Legend, Table Legends 1-5 from The Transcription Factor ZNF217 Is a Prognostic Biomarker and Therapeutic Target during Breast Cancer Progression

15. Supplementary Table S1 from Exon-Level Microarray Analyses Identify Alternative Splicing Programs in Breast Cancer

16. Data from Lrig1 Is an Estrogen-Regulated Growth Suppressor and Correlates with Longer Relapse-Free Survival in ERα-Positive Breast Cancer

17. Supplementary Materials and Methods from The Transcription Factor ZNF217 Is a Prognostic Biomarker and Therapeutic Target during Breast Cancer Progression

18. Supplementary Table 1 from The Transcription Factor ZNF217 Is a Prognostic Biomarker and Therapeutic Target during Breast Cancer Progression

21. Supplementary Figures S1-S7 from Targeted Treatment of Metastatic Breast Cancer by PLK1 siRNA Delivered by an Antioxidant Nanoparticle Platform

23. Supplementary Figure 5 from A Central Role for RAF→MEK→ERK Signaling in the Genesis of Pancreatic Ductal Adenocarcinoma

24. Supplementary Figure 1 from Temporal Dissection of Tumorigenesis in Primary Cancers

25. Supplementary Figure 2 from Temporal Dissection of Tumorigenesis in Primary Cancers

26. Supplementary Figure Legends 1-4, Methods from Temporal Dissection of Tumorigenesis in Primary Cancers

29. Supplementary Table 2 from Temporal Dissection of Tumorigenesis in Primary Cancers

32. Supplementary Figures S1-S3 from Pathway-Enriched Gene Signature Associated with 53BP1 Response to PARP Inhibition in Triple-Negative Breast Cancer

35. Supplementary Figure 3 from Temporal Dissection of Tumorigenesis in Primary Cancers

37. Supplementary Table 5 from The Transcription Factor ZNF217 Is a Prognostic Biomarker and Therapeutic Target during Breast Cancer Progression

38. Supplementary Data from Exon-Level Microarray Analyses Identify Alternative Splicing Programs in Breast Cancer

40. Supplementary Table 2 from A Central Role for RAF→MEK→ERK Signaling in the Genesis of Pancreatic Ductal Adenocarcinoma

43. Supplementary Figure 4 from Temporal Dissection of Tumorigenesis in Primary Cancers

44. Supplementary Table 2 from The Transcription Factor ZNF217 Is a Prognostic Biomarker and Therapeutic Target during Breast Cancer Progression

46. Supplementary Figures S4-S12 from Pathway-Enriched Gene Signature Associated with 53BP1 Response to PARP Inhibition in Triple-Negative Breast Cancer

47. Supplementary Tables S1-S4 from Pathway-Enriched Gene Signature Associated with 53BP1 Response to PARP Inhibition in Triple-Negative Breast Cancer

48. Supplementary Table 1 from Temporal Dissection of Tumorigenesis in Primary Cancers

49. Supplementary Table 3 from The Transcription Factor ZNF217 Is a Prognostic Biomarker and Therapeutic Target during Breast Cancer Progression

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