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146 results on '"Hattersley, Andrew T."'

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1. A multi-layer functional genomic analysis to understand noncoding genetic variation in lipids

2. Genetic effects on the timing of parturition and links to fetal birth weight

3. Infancy-onset diabetes caused by de-regulated AMPylation of the human endoplasmic reticulum chaperone BiP

4. Multi-ancestry genome-wide study in >2.5 million individuals reveals heterogeneity in mechanistic pathways of type 2 diabetes and complications

5. Infancy‐onset diabetes caused by de‐regulated AMPylation of the human endoplasmic reticulum chaperone BiP

6. Additional file 26 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

7. Additional file 34 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

8. Additional file 24 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

9. Additional file 16 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

10. Author Correction : The power of genetic diversity in genome-wide association studies of lipids

11. Additional file 11 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

12. Additional file 1 of Comparison of causal forest and regression-based approaches to evaluate treatment effect heterogeneity: an application for type 2 diabetes precision medicine

13. Additional file 31 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

14. Additional file 15 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

15. Additional file 31 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

16. Additional file 6 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

17. Additional file 9 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

18. Additional file 3 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

19. Additional file 24 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

20. Additional file 3 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

21. Additional file 14 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

22. Additional file 26 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

23. Additional file 6 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

24. Additional file 13 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

25. Type 2 Diabetes risk alleles in Peptidyl-glycine Alpha-amidating Monooxygenase influence GLP-1 levels and response to GLP-1 Receptor Agonists

26. Additional file 9 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

27. Additional file 34 of Implicating genes, pleiotropy, and sexual dimorphism at blood lipid loci through multi-ancestry meta-analysis

28. A saturated map of common genetic variants associated with human height

29. Identification of GCK-MODY in cases of neonatal hyperglycemia: a case series and review of clinical features

30. Multi-ancestry genetic study of type 2 diabetes highlights the power of diverse populations for discovery and translation

31. Non-coding variants disrupting a tissue-specific regulatory element in HK1 cause congenital hyperinsulinism

33. Genetics of early-life head circumference and genetic correlations with neurological, psychiatric and cognitive outcomes

35. The power of genetic diversity in genome-wide association studies of lipids

36. Reappearance of C-Peptide During the Third Trimester of Pregnancy in Type 1 Diabetes: Pancreatic Regeneration or Fetal Hyperinsulinism?

37. Evaluating associations between the benefits and risks of drug therapy in type 2 diabetes: a joint modeling approach

38. Molecular reductions in glucokinase activity increase counter-regulatory responses to hypoglycemia in mice and humans with diabetes

39. Additional file 5 of Choice of HbA1c threshold for identifying individuals at high risk of type 2 diabetes and implications for diabetes prevention programmes: a cohort study

40. Additional file 3 of Choice of HbA1c threshold for identifying individuals at high risk of type 2 diabetes and implications for diabetes prevention programmes: a cohort study

42. The power of genetic diversity in genome-wide association studies of lipids

43. Extended data Wellcome Open Research 16097.pdf

44. Loss of MANF causes childhood-onset syndromic diabetes due to increased endoplasmic reticulum stress

45. Extended data Wellcome Open Research 12688.pdf

46. Additional file 4 of Choice of HbA1c threshold for identifying individuals at high risk of type 2 diabetes and implications for diabetes prevention programmes: a cohort study

47. Risk factors for genital infections in people initiating SGLT2 inhibitors and their impact on discontinuation

48. Hyperglycemia and Adverse Pregnancy Outcome (HAPO) study: common genetic variants in GCK and TCF7L2 are associated with fasting and postchallenge glucose levels in pregnancy and with the new consensus definition of gestational diabetes mellitus from the International Association of Diabetes and Pregnancy Study Groups

49. Additional file 1 of Whole blood co-expression modules associate with metabolic traits and type 2 diabetes: an IMI-DIRECT study

50. Additional file 2 of Whole blood co-expression modules associate with metabolic traits and type 2 diabetes: an IMI-DIRECT study

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