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12 results on '"Antenucci M"'

1. Effects of 1,4-butanediol dimethacrylate and urethane dimethacrylate on HL-60 cell metabolism

Author

Nocca G., Martorana G., De Sole P., De Palma F., Callà C., Corsale P., Antenucci M., Gambarini G., Chimenti C., Giardina B., and Lupi A.

Abstract

The polymerization of methacrylic monomers present in dental composite resins never reaches completion and therefore the leakage of residual monomers into the oral cavity and into biological fluids can cause local and systemic adverse effects. This work was carried out to study the in vitro biochemical interactions of urethane dimethacrylate and 1,4-butanediol dimethacrylate monomers with HL-60 cells, a cell line assumed as an experimental model for simulating granulocyte behaviour. Our main finding was that both monomers induce cell differentiation at toxic concentrations and that cytotoxicity seems to be caused by alterations of glucose metabolism arising from mitochondrial dysfunction rather than from oxidative stress, which could not be altogether verified under our experimental conditions. Our study could be considered as a useful approach to investigate the biochemical mechanisms that contribute to the cytotoxicity of methacrylate compounds and it underlines the importance of assessing such parameters for testing biocompatibility in order to promote the development of better and safer dental materials.

Published
2009

4. Quando el tempo està suspendido: individuo y familia en la anorexia mental

Author

Onnis, Luigi, Antenucci, M., Benedetti, P., Bernardini, M., D'Amore, S., Dentale, R. C., Di Gregorio, L., Ferilli, C., Giannuzzi, M., Giovannetti, D., Maurelli, F., Mulè, A. M., Squitieri, A., and Tarantino, F.

Subjects
Eating Disorders, Individual Patient, Culture Family, Suspended Time
Published
2005

10. VITATOPS, the VITAmins TO Prevent Stroke trial: rationale and design of a randomised trial of B-vitamin therapy in patients with recent transient ischaemic attack or stroke (NCT00097669) (ISRCTN74743444)

Author

Graeme Hankey, Baker, R. I., Eikelboom, J. W., Gelavis, A., Hickling, S. C., Jamrozik, K., Bockxmeer, F. M., Vasikaran, S., Algra, A., Chen, C., Wong, M. C., Cheung, R., Wong, L., Divjak, I., Ferro, J., Gommans, J., Groppa, S., Hill, M., Spence, D., Lees, K., Lisheng, L., Navarro, J., Ranawaka, U., Ricci, S., Schmidt, R., Slivka, A., Tan, K., Tsiskaridze, A., Uddin, W., Vanhooren, G., Xavier, D., Armitage, J., Hobbs, M., Le, M., Sudlow, C., Wheatley, K., Yi, Q., Bulder, M., Ho, W. K., Klijn, K., Koedam, E., Langton, P., Nijboer, E., Tuch, P., Pizzi, J., Tang, M., Antenucci, M., Chew, Y., Chinnery, D., Cockayne, C., Loh, K., Mcmullin, L., Smith, F., Freitas, G., Song, S., and Vitatops, Trial Study Grp

11. G6PD Murcia, G6PD Ube and G6PD Orissa: report of three G6PD mutations unusual for Italian population

Author

Bruno Giardina, Ettore Capoluongo, Cecilia Zuppi, Mirca Antenucci, Angelo Minucci, Minucci, A, Antenucci, M, Giardina, B, Zuppi, C, and Capoluongo, E

Subjects
Adult, Male, Hemolytic anemia, Clinical Biochemistry, Glucosephosphate Dehydrogenase, Biology, Genetic Heterogeneity, Settore BIO/12 - BIOCHIMICA CLINICA E BIOLOGIA MOLECOLARE CLINICA, Ethnicity, medicine, Humans, Family, G6PD ORISSA, Gene, chemistry.chemical_classification, Genetics, Infant, Newborn, G6PD MUTATION, General Medicine, medicine.disease, Italian population, GENE, Glucosephosphate Dehydrogenase Deficiency, Enzyme, Italy, chemistry, Biochemistry, Mutation, Mutation (genetic algorithm), Female
Published
2010

12. Description of a novel missense mutation of glucose-6-phosphate dehydrogenase gene associated with asymptomatic high enzyme deficiency

Author

Paola Concolino, Ettore Capoluongo, Cecilia Zuppi, Angelo Minucci, Concetta Santonocito, Bruno Giardina, Mirca Antenucci, Franco Ameglio, Minucci, A, Concolino, P, Antenucci, M, Santonocito, C, Ameglio, F, Zuppi, C, Giardina, B, and Capoluongo, E.

Subjects
Adult, Male, congenital, hereditary, and neonatal diseases and abnormalities, Clinical Biochemistry, DNA Mutational Analysis, Molecular Sequence Data, Mutation, Missense, Dehydrogenase, Biology, Glucosephosphate Dehydrogenase, medicine.disease_cause, Asymptomatic, Exon, In vivo, hemic and lymphatic diseases, parasitic diseases, medicine, Glucose-6-Phosphate Dehydrogenase Gene, Missense mutation, Humans, Genetics, Mutation, Molecular epidemiology, Base Sequence, nutritional and metabolic diseases, General Medicine, Molecular biology, Glucosephosphate Dehydrogenase Deficiency, Case-Control Studies, medicine.symptom
Abstract

We report a case of an asymptomatic young subject affected by severe deficiency of Glucose 6-phosphate dehydrogenase (G6PD) activity. A novel genetic mutation (G130A) in the third exon was found. We named this novel mutation the “G6PD RIGNANO variant”. These findings may contribute to a better knowledge of molecular epidemiology of the G6PD mutation and may represent an additional variant to be studied for a deep comprehension of in vivo compensation mechanisms of G6PD deficiency.

Published
2007

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