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17 results on '"van Beers E"'

1. Ektacytometry Analysis of Post-splenectomy Red Blood Cell Properties Identifies Cell Membrane Stability Test as a Novel Biomarker of Membrane Health in Hereditary Spherocytosis

Author

Opleiding Neurologie, CDL Rode cel, Centraal Diagnostisch Laboratorium, Zorgeenheid Kinderchirurgie Medisch, Child Health, Poli Van Creveldkliniek Medisch, Haematologie patientenzorg, CDL Arcadia, Circulatory Health, Unit Opleiding Aios, Berrevoets, M C, Bos, J, Huisjes, R, Merkx, T H, van Oirschot, B A, van Solinge, W W, Verweij, J W, Lindeboom, M Y A, van Beers, E J, Bartels, M, van Wijk, R, Rab, M A E, Opleiding Neurologie, CDL Rode cel, Centraal Diagnostisch Laboratorium, Zorgeenheid Kinderchirurgie Medisch, Child Health, Poli Van Creveldkliniek Medisch, Haematologie patientenzorg, CDL Arcadia, Circulatory Health, Unit Opleiding Aios, Berrevoets, M C, Bos, J, Huisjes, R, Merkx, T H, van Oirschot, B A, van Solinge, W W, Verweij, J W, Lindeboom, M Y A, van Beers, E J, Bartels, M, van Wijk, R, and Rab, M A E

Published
2021

2. Predictors of short-term and long-term mortality in critically ill patients admitted to the intensive care unit following allogeneic stem cell transplantation

Author

van der Heiden, P. L. J., Arbous, M. S., van Beers, E. J., van den Bergh, W. M., le Cessie, S., Demandt, A. M. P., Eefting, M., Hess, C., Kusadasi, N., Marijt, W. A. F., van Mook, W. N. K. A., Mueller, M. C. A., Tuinman, P. R., van Vliet, M., van Westerloo, D. J., Blijlevens, N. M. A., van der Heiden, P. L. J., Arbous, M. S., van Beers, E. J., van den Bergh, W. M., le Cessie, S., Demandt, A. M. P., Eefting, M., Hess, C., Kusadasi, N., Marijt, W. A. F., van Mook, W. N. K. A., Mueller, M. C. A., Tuinman, P. R., van Vliet, M., van Westerloo, D. J., and Blijlevens, N. M. A.

Published
2019

3. Predictors of short-term and long-term mortality in critically ill patients admitted to the intensive care unit following allogeneic stem cell transplantation

Author

van der Heiden, P. L. J., Arbous, M. S., van Beers, E. J., van den Bergh, W. M., le Cessie, S., Demandt, A. M. P., Eefting, M., Hess, C., Kusadasi, N., Marijt, W. A. F., van Mook, W. N. K. A., Mueller, M. C. A., Tuinman, P. R., van Vliet, M., van Westerloo, D. J., Blijlevens, N. M. A., van der Heiden, P. L. J., Arbous, M. S., van Beers, E. J., van den Bergh, W. M., le Cessie, S., Demandt, A. M. P., Eefting, M., Hess, C., Kusadasi, N., Marijt, W. A. F., van Mook, W. N. K. A., Mueller, M. C. A., Tuinman, P. R., van Vliet, M., van Westerloo, D. J., and Blijlevens, N. M. A.

Published
2019

4. Predictors of short-term and long-term mortality in critically ill patients admitted to the intensive care unit following allogeneic stem cell transplantation

Author

van der Heiden, P. L. J., Arbous, M. S., van Beers, E. J., van den Bergh, W. M., le Cessie, S., Demandt, A. M. P., Eefting, M., Hess, C., Kusadasi, N., Marijt, W. A. F., van Mook, W. N. K. A., Mueller, M. C. A., Tuinman, P. R., van Vliet, M., van Westerloo, D. J., Blijlevens, N. M. A., van der Heiden, P. L. J., Arbous, M. S., van Beers, E. J., van den Bergh, W. M., le Cessie, S., Demandt, A. M. P., Eefting, M., Hess, C., Kusadasi, N., Marijt, W. A. F., van Mook, W. N. K. A., Mueller, M. C. A., Tuinman, P. R., van Vliet, M., van Westerloo, D. J., and Blijlevens, N. M. A.

Published
2019

5. Predictors of short-term and long-term mortality in critically ill patients admitted to the intensive care unit following allogeneic stem cell transplantation

Author

Poli Van Creveldkliniek Medisch, van der Heiden, P. L. J., Arbous, M. S., van Beers, E. J., van den Bergh, W. M., le Cessie, S., Demandt, A. M. P., Eefting, M., Hess, C., Kusadasi, N., Marijt, W. A. F., van Mook, W. N. K. A., Mueller, M. C. A., Tuinman, P. R., van Vliet, M., van Westerloo, D. J., Blijlevens, N. M. A., Poli Van Creveldkliniek Medisch, van der Heiden, P. L. J., Arbous, M. S., van Beers, E. J., van den Bergh, W. M., le Cessie, S., Demandt, A. M. P., Eefting, M., Hess, C., Kusadasi, N., Marijt, W. A. F., van Mook, W. N. K. A., Mueller, M. C. A., Tuinman, P. R., van Vliet, M., van Westerloo, D. J., and Blijlevens, N. M. A.

Published
2019

9. Screening for hemosiderosis in patients receiving multiple red blood cell transfusions

Author

Poli Van Creveldkliniek Medisch, CDL Staf Patiëntenzorg KC, Other research (not in main researchprogram), Circulatory Health, de Jongh, Adriaan D, van Beers, E J, de Vooght, K M K, Schutgens, R E G, Poli Van Creveldkliniek Medisch, CDL Staf Patiëntenzorg KC, Other research (not in main researchprogram), Circulatory Health, de Jongh, Adriaan D, van Beers, E J, de Vooght, K M K, and Schutgens, R E G

Published
2017

14. Cross-species comparison of aCGH data from mouse and human BRCA1- and BRCA2-mutated breast cancers

Author

Holstege, H. (author), Van Beers, E. (author), Velds, A. (author), Liu, X. (author), Joosse, S.A. (author), Klarenbeek, S. (author), Schut, E. (author), Kerkhoven, R. (author), Klijn, C.N. (author), Wessels, L.F.A. (author), Nederlof, P.M. (author), Jonkers, J. (author), Holstege, H. (author), Van Beers, E. (author), Velds, A. (author), Liu, X. (author), Joosse, S.A. (author), Klarenbeek, S. (author), Schut, E. (author), Kerkhoven, R. (author), Klijn, C.N. (author), Wessels, L.F.A. (author), Nederlof, P.M. (author), and Jonkers, J. (author)

Abstract

Background: Genomic gains and losses are a result of genomic instability in many types of cancers. BRCA1- and BRCA2-mutated breast cancers are associated with increased amounts of chromosomal aberrations, presumably due their functions in genome repair. Some of these genomic aberrations may harbor genes whose absence or overexpression may give rise to cellular growth advantage. So far, it has not been easy to identify the driver genes underlying gains and losses. A powerful approach to identify these driver genes could be a cross-species comparison of array comparative genomic hybridization (aCGH) data from cognate mouse and human tumors. Orthologous regions of mouse and human tumors that are commonly gained or lost might represent essential genomic regions selected for gain or loss during tumor development. Methods: To identify genomic regions that are associated with BRCA1- and BRCA2-mutated breast cancers we compared aCGH data from 130 mouse Brca1?/?;p53?/?, Brca2?/?;p53?/? and p53?/? mammary tumor groups with 103 human BRCA1-mutated, BRCA2-mutated and non-hereditary breast cancers. Results: Our genome-wide cross-species analysis yielded a complete collection of loci and genes that are commonly gained or lost in mouse and human breast cancer. Principal common CNAs were the well known MYCassociated gain and RB1/INTS6-associated loss that occurred in all mouse and human tumor groups, and the AURKA-associated gain occurred in BRCA2-related tumors from both species. However, there were also important differences between tumor profiles of both species, such as the prominent gain on chromosome 10 in mouse Brca2?/?;p53?/? tumors and the PIK3CA associated 3q gain in human BRCA1-mutated tumors, which occurred in tumors from one species but not in tumors from the other species. This disparity in recurrent aberrations in mouse and human tumors might be due to differences in tumor cell type or genomic organization between both species. Conclusions: The selection of the oncog, Mediamatics, Electrical Engineering, Mathematics and Computer Science

Published
2010
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15. Cross-species comparison of aCGH data from mouse and human BRCA1- and BRCA2-mutated breast cancers

Author

Holstege, H. (author), Van Beers, E. (author), Velds, A. (author), Liu, X. (author), Joosse, S.A. (author), Klarenbeek, S. (author), Schut, E. (author), Kerkhoven, R. (author), Klijn, C.N. (author), Wessels, L.F.A. (author), Nederlof, P.M. (author), Jonkers, J. (author), Holstege, H. (author), Van Beers, E. (author), Velds, A. (author), Liu, X. (author), Joosse, S.A. (author), Klarenbeek, S. (author), Schut, E. (author), Kerkhoven, R. (author), Klijn, C.N. (author), Wessels, L.F.A. (author), Nederlof, P.M. (author), and Jonkers, J. (author)

Abstract

Background: Genomic gains and losses are a result of genomic instability in many types of cancers. BRCA1- and BRCA2-mutated breast cancers are associated with increased amounts of chromosomal aberrations, presumably due their functions in genome repair. Some of these genomic aberrations may harbor genes whose absence or overexpression may give rise to cellular growth advantage. So far, it has not been easy to identify the driver genes underlying gains and losses. A powerful approach to identify these driver genes could be a cross-species comparison of array comparative genomic hybridization (aCGH) data from cognate mouse and human tumors. Orthologous regions of mouse and human tumors that are commonly gained or lost might represent essential genomic regions selected for gain or loss during tumor development. Methods: To identify genomic regions that are associated with BRCA1- and BRCA2-mutated breast cancers we compared aCGH data from 130 mouse Brca1?/?;p53?/?, Brca2?/?;p53?/? and p53?/? mammary tumor groups with 103 human BRCA1-mutated, BRCA2-mutated and non-hereditary breast cancers. Results: Our genome-wide cross-species analysis yielded a complete collection of loci and genes that are commonly gained or lost in mouse and human breast cancer. Principal common CNAs were the well known MYCassociated gain and RB1/INTS6-associated loss that occurred in all mouse and human tumor groups, and the AURKA-associated gain occurred in BRCA2-related tumors from both species. However, there were also important differences between tumor profiles of both species, such as the prominent gain on chromosome 10 in mouse Brca2?/?;p53?/? tumors and the PIK3CA associated 3q gain in human BRCA1-mutated tumors, which occurred in tumors from one species but not in tumors from the other species. This disparity in recurrent aberrations in mouse and human tumors might be due to differences in tumor cell type or genomic organization between both species. Conclusions: The selection of the oncog, Mediamatics, Electrical Engineering, Mathematics and Computer Science

Published
2010
Full Text
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16. Discovering genetic profiles by array-CGH in familial breast tumors

Author

Nederlof, P.M. (author), Van Beers, E. (author), Joosse, S. (author), Hogervorst, F.B.L. (author), Wessels, L.F.A. (author), Devilee, P. (author), Cornelisse, C. (author), Oldenburg, R. (author), Verhoef, S. (author), Van 't Veer, L.J. (author), Nederlof, P.M. (author), Van Beers, E. (author), Joosse, S. (author), Hogervorst, F.B.L. (author), Wessels, L.F.A. (author), Devilee, P. (author), Cornelisse, C. (author), Oldenburg, R. (author), Verhoef, S. (author), and Van 't Veer, L.J. (author)

Published
2005

17. Discovering genetic profiles by array-CGH in familial breast tumors

Author

Nederlof, P.M. (author), Van Beers, E. (author), Joosse, S. (author), Hogervorst, F.B.L. (author), Wessels, L.F.A. (author), Devilee, P. (author), Cornelisse, C. (author), Oldenburg, R. (author), Verhoef, S. (author), Van 't Veer, L.J. (author), Nederlof, P.M. (author), Van Beers, E. (author), Joosse, S. (author), Hogervorst, F.B.L. (author), Wessels, L.F.A. (author), Devilee, P. (author), Cornelisse, C. (author), Oldenburg, R. (author), Verhoef, S. (author), and Van 't Veer, L.J. (author)

Published
2005

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