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22 results on '"Schinke, Thorsten"'

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1. Early enzyme replacement therapy prevents dental and craniofacial abnormalities in a mouse model of mucopolysaccharidosis type VI

2. The WNT1(G177C) mutation specifically affects skeletal integrity in a mouse model of osteogenesis imperfecta type XV

3. Decreased Trabecular Bone Mass in Col22a1-Deficient Mice

4. The WNT1(G177C) mutation specifically affects skeletal integrity in a mouse model of osteogenesis imperfecta type XV

5. Decreased Trabecular Bone Mass in Col22a1-Deficient Mice

6. TFEB deficiency attenuates mitochondrial degradation upon brown adipose tissue whitening at thermoneutrality

7. Arylsulfatase K inactivation causes mucopolysaccharidosis due to deficient glucuronate desulfation of heparan and chondroitin sulfate

8. Arylsulfatase K inactivation causes mucopolysaccharidosis due to deficient glucuronate desulfation of heparan and chondroitin sulfate

9. Arylsulfatase K inactivation causes mucopolysaccharidosis due to deficient glucuronate desulfation of heparan and chondroitin sulfate

10. Arylsulfatase K inactivation causes mucopolysaccharidosis due to deficient glucuronate desulfation of heparan and chondroitin sulfate

11. The Lysosomal Protein Arylsulfatase B Is a Key Enzyme Involved in Skeletal Turnover

12. The Lysosomal Protein Arylsulfatase B Is a Key Enzyme Involved in Skeletal Turnover

13. The chaperone activity of 4PBA ameliorates the skeletal phenotype of Chihuahua, a zebrafish model for dominant osteogenesis imperfecta

14. Post-weaning epiphysiolysis causes distal femur dysplasia and foreshortened hindlimbs in fetuin-A-deficient mice

15. The chaperone activity of 4PBA ameliorates the skeletal phenotype of Chihuahua, a zebrafish model for dominant osteogenesis imperfecta

16. Post-weaning epiphysiolysis causes distal femur dysplasia and foreshortened hindlimbs in fetuin-A-deficient mice

17. Parathyroid hormone induces expression and proteolytic processing of Rankl in primary murine osteoblasts

18. Parathyroid hormone induces expression and proteolytic processing of Rankl in primary murine osteoblasts

19. The clock genes Period 2 and Cryptochrome 2 differentially balance bone formation

20. Glucocorticoids suppress bone formation by attenuating osteoblast differentiation via the monomeric glucocorticoid receptor

21. the clock genes Period 2 and Cryptochrome 2 differentially balance bone formation

22. the clock genes Period 2 and Cryptochrome 2 differentially balance bone formation

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