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1. Templated misfolding of TAU by prion-like seeding along neuronal circuits impairs synaptic and cognitive function in TAU transgenic mice

2. Activation of LXR/RXR receptors increases cholesterol homeostasis and neuronal activity in primary cultures of neurons expressing or not human APP

3. Induction of HMGCR dependent cholesterol biosynthesis is required to improve synaptic plasticity driven by LXR/RXR activation-mediated cholesterol efflux

4. AMYLOID-INDUCED TAUOPATHY CONTRIBUTES TO SYNAPTIC AND COGNITIVE DEFICITS IN A TRANSGENIC MODEL FOR ALZHEIMER’S DISEASE

5. Activation of cholesterol turnover as a pharmacological approach to increase neuronal activity in Alzheimer's disease

6. AMYLOID PRECURSOR PROTEIN REGULATES NEURONAL CHOLESTEROLTURNOVER NEEDED FOR SYNAPTIC ACTIVITY

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