18 results on '"Mittler, Robert S."'
Search Results
2. Multivalent 4-1BB binding aptamers costimulate CD8+ T cells and inhibit tumor growth in mice
3. Immune suppression or enhancement by CD137 T cell costimulation during acute viral infection is time dependent
4. Modulating Protective and Pathogenic CD4+ Subsets via CD137 in Type 1 Diabetes
5. CD137-mediated immunotherapy for allergic asthma
6. Combined CD137 (4-1BB) and adjuvant therapy generates a developing pool of peptide-specific CD8 memory T cells
7. Vaccination with dendritic cells pulsed with apoptotic tumors in combination with anti-OX40 and anti-4-1BB monoclonal antibodies induces T cell–mediated protective immunity in Her-2/neu transgenic mice
8. Engagement of the CD137 (4-1BB) costimulatory molecule inhibits and reverses the autoimmune process in collagen-induced arthritis and establishes lasting disease resistance
9. 4-1BB and OX40 stimulation enhance CD8 and CD4 T-cell responses to a DNA prime, poxvirus boost vaccine
10. Anti-CD137 Antibodies in the Treatment of Autoimmune Disease and Cancer
11. Role of 4-1BB in Allograft Rejection Mediated by CD8+ T Cells
12. CD137 costimulatory T cell receptor engagement reverses acute disease in lupus-prone NZB × NZW F1 mice
13. CD137-Mediated T Cell Co-Stimulation Terminates Existing Autoimmune Disease in SLE-Prone NZB/NZW F1 Mice
14. 4-1BB co-stimulation enhances human CD8+ T cell priming by augmenting the proliferation and survival of effector CD8+ T cells
15. Anti-4-1BB Monoclonal Antibodies Abrogate T Cell-dependent Humoral Immune Responses In Vivo through the Induction of Helper T Cell Anergy
16. 4-1BB Costimulatory Signals Preferentially Induce CD8+ T Cell Proliferation and Lead to the Amplification In Vivo of Cytotoxic T Cell Responses
17. Surface Expression of CD28 Single Chain Fv for Costimulation by Tumor Cells
18. The Two Membrane Proximal Domains of CD4 Interact with the T Cell Receptor
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.