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The remote sensing satellite is developing toward high resolution, large data capacity, and fast transmission rate. The ground receiver is correspondingly required to have the high parallel to improve real-time reception and processing capabilities. In the frequency domain, the parallel processing of multiple subbands is achieved by dividing broadband into narrow bands. However, narrow-band subbands are reconstructed into broadband signals, which can lead to cross-subband distortion. It will affect the accuracy of high-resolution remote sensing images. In this article, the subband division and reconstruction framework is proposed by combining the analog filter with the digital filter. The phase calibration methods and digital filter optimization are proposed to improve the amplitude and phase consistency of the reconstructed signal. The simulation results show that the proposed amplitude consistency optimization method effectively reduces the reconstruction error within 0.001 dB. The proposed phase calibration method effectively reduces the bit error rate of the reconstructed signal. The maximum deviation is no more than 0.1%. Experiments have shown that the optimization method can reduce the distortion error of high-resolution remote sensing images.

The large-scale application of flexible DC power transmission technology leads to DC bias in transformer cores, and the vibration analysis of transformers under the influence of DC bias is becoming a hot topic. In this paper, based on the "electromagnetic-mechanical" coupling model, we propose a bias vibration analysis method for large power transformers. The electromagnetic forces calculated in the electromagnetic field are mapped to the surfaces of structural members and tanks in the structural force field by means of a grid mapping method, facilitating the direct calculation of transformer vibration displacements under DC bias conditions. Finally, based on the coupled magnetic-structural analysis, the maximum deformation of different structures of the transformer under different DC bias magnetism is studied. The vibration response band of the transformer is above 100Hz, where the DC input of 25A per phase causes the maximum displacement deformation of 39.06mm in the transformer tank, which provides the analysis basis for the safe operation of the transformer.

Abstract The adaptive interference cancellation technique has been widely utilized in radar, GPS, data link, etc., systems to address challenges from external interference, such as co-site and hostile interference. Since the anti-jamming performance of the adaptive interference cancellation technique is sensitive to group delay mismatch between channels, the group delay mismatch becomes one of the main factors that limit the system’s anti-jamming capability. However, the traditional adaptive interference cancellation system’s mathematical model cannot quantitatively characterize the group delay mismatch effect on the wideband interference cancellation performance. In this paper, the mathematical model of the wideband adaptive spatial interference cancellation (ASIC) system is established, which considers the group delay mismatch, to quantitatively analyze the impact of group delay mismatch on the hostile interference cancellation. The mathematical model utilizes the weighted multi-tone signals to fit the wideband interference, and then, delay differences are attached to each tone signal to simulate the group delay mismatch. Then, the analytic expressions of weight and interference cancellation ratio are derived, which consider the interference bandwidth and group delay mismatch, to quantitatively analyze the group delay mismatch effect on the anti-jamming performance of the wideband ASIC system. Simulation results indicate that the theoretical analysis based on the mathematical model of wideband ASIC system are accurate, which can achieve the quantitative analysis of the group delay mismatch effect on the WIC performance.

This paper takes the BKFSZT-30000/400 three-phase eight-limb magnetically controlled shunt reactor as the research object. Firstly, an electromagnetic transient simulation model is established based on the parity principle to verify the operating characteristics of the MCSR during direct energization and pre-excitation energization. On this basis, the influence of the pre-excitation capacity and the target capacity on the output characteristics of the MCSR is investigated. The results show that during the pre-excitation process the currents of working and control winding produce a DC component and a harmonic component dominated by the second harmonic, with the size of both DC components mainly influenced by the pre-excitation capacity and the attenuation determined by MCSR; the variation of the working harmonics is mainly influenced by the target capacity, while the size and attenuation of the control harmonics are determined by the pre-excitation capacity and the target capacity respectively. The results of the study clarify the output characteristics of the MCSR when energization with pre-excitation and provide a reference for further analysis of the interaction with the grid. Finally, to solve the problem of no reliable external power supply for pre-excitation, a fast excitation energization method with short-connected of the compensation winding is provided and verified by simulation.

Permanent magnet synchronous motors are widely used in various fields for the advantages of high power density, high efficiency and high reliability. This paper analyzes the cogging torque of generators with different slot and pole coordination, and selects the optimal pole–slot ratio. Then, this paper analyzes the structural differences between the outer rotor and the inner rotor of permanent magnet synchronous motor, and selects the structural form of the outer rotor for analysis. Based on the electromagnetic design parameters of the motor, a two-dimensional finite element model is established in Maxwell to simulate and analyze the performance at no-load, rated load and short-circuit. The internal magnetic field, the distortion rate of voltage waveform, torque and short circuit current of the motor are studied, and the demagnetization of the permanent magnet under the worst operating condition is investigated. The simulation results show that the design scheme meets the design objectives.

The interference cancellation ratio (ICR) is a key performance indicator of digital-to-analog hybrid radio frequency (RF) interference cancellation systems. Aiming at the low convergence speed of a digital-to-analog hybrid RF interference cancellation system based on a multi-tap structure (MDARFICS), a novel, fast, and robust variable-step-size least-mean-square (LMS) algorithm based on an improved hyperbolic tangent function (IHVSS-LMS) is proposed. The IHVSS-LMS algorithm adopts an improved hyperbolic tangent function and uses adjustable parameters and the iteration number to jointly adjust the step size, which improves the convergence speed and reduces the computational complexity. Moreover, by using the prior information of the input signal, the non-linear relationship between the step size and the input signal power is established, which enhances the robustness and the ability to suppress interference with mutable power. The IHVSS-LMS algorithm is applied to the MDARFICS. Through theoretical derivation, the convergence speed and the steady-state expressions of the interference cancellation ratio of the MDARFICS are obtained. The simulation results show that under the conditions of high and low signal-to-noise ratio (SNR), the robustness, convergence speed, and steady-state error performance of the IHVSS-LMS algorithm are better than the existing variable-step-size algorithm. The experimental results show that using the IHVSS-LMS algorithm, the MDARFICS can not only effectively accelerate the convergence speed by at least three times but can also improve the ICR by more than 3 dB.

Background/Aims: MicroRNAs (miRNAs) are a group of non-coding RNAs that play diverse roles in pancreatic carcinogenesis. In pancreatic ductal adenocarcinoma (PDAC), NF-kB is constitutively activated in most patients and is linked to a mutation in KRAS via IkB kinase complex 1 (IKK1, also known as IKKa). We investigated the link between PDAC aggressiveness and miR-1290. Methods: We used miRCURYTM LNA Array and in situ hybridization to investigate candidate miRNAs and validated the findings with PCR. The malignant behavior of cell lines was assessed with Cell Counting Kit-8, colony formation, and Transwell assays. A dual-luciferase reporter assay was used to evaluate the interaction between miR-1290 and IKK1. Protein expression was observed by western blotting. Results: In this study, 36 miRNAs were dysregulated in high-grade pancreatic intraepithelial neoplasia (PanIN) and PDAC tissues compared with low-grade PanIN tissues. The area under the curve values of miR-1290 and miR-31-5p were 0.829 and 0.848, respectively (95% confidence interval, 0.722–0.936 and 0.749–0.948, both P < 0.001). There was a significant correlation between miR-1290 and histological differentiation (P = 0.029), pT stage (P = 0.006), and lymph node metastasis (P = 0.001). In addition, the in vitro work showed that miR-1290 promoted PDAC cell proliferation, invasion, and migration. Western blotting and the dual-luciferase reporter assay showed that miR-1290 promoted cancer aggressiveness by directly targeting IKK1. The synergist effect of miR-1290 on the proliferation and metastasis of PDAC cells was attenuated and enhanced by IKK1 overexpression and knockdown, respectively. Consistent with the in vitro results, a subcutaneous tumor mouse model showed that miR-1290 functioned as a potent promoter of PDAC in vivo. Conclusion: MiR-1290 may act as an oncogene by directly targeting the 3’-untranslated region of IKK1, and the miR-1290/IKK1 pathway may prove to be a novel diagnostic and therapeutic target for PDAC.

The long non-coding RNA maternally expressed gene 3 (MEG3) is frequently dysregulated in human cancers; however, its roles in colorectal cancer (CRC) development are largely unknown. Here, we reported that MEG3 was down-regulated in CRC tissues and CRC patients with lower MEG3 showed poorer overall survival and disease-free survival than those with higher MEG3 level. MEG3 over-expression represses CRC cells proliferation and migration in vivo and in vitro, while MEG3 knockdown leads to the enhanced proliferation and metastasis of CRC cells. In CRC cells, MEG3 over-expression is related to decreased Clusterin mRNA and the corresponding protein levels, and it also directly binds to Clusterin protein through its 732–1174 region. In further, Clusterin over-expression rescues the compromised abilities of proliferation and metastasis induced by MEG3 over-expression, suggesting that MEG3 inhibits the CRC progression through regulating the Clusterin activities. Additionally, we found that 1α,25-(OH)2D and vitamin D receptor (VDR) stimulate MEG3 expression in CRC cells through directly binding to its promoter. These results suggested that MEG3 functions as a tumor suppressor in CRC via regulating the Clusterin activities and may underlie the anticancer activities of vitamin D on CRC cells. The VDR/MEG3/Clusterin signaling pathway may serve as potential therapeutic targets and prognosis biomarkers for CRC patients in future. Keywords: lncRNA, CRC, MEG3, Clusterin, Vitamin D