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One of the major problems faced by modern society is to find a suitable disposal for excess solid waste generated, particularly disposable products. In this context, the use of recycled PET as reinforcement in composites, is an idea that arises in order to seek alternatives for the recycling of this waste. We studied the influence of the variation in the mechanical properties of PET fiber reinforced polyester. Test specimens were made of pure matrix and 5 to 10% of PET in the particle size of 40, 60 and 80 mesh. We evaluated the mechanical behavior of composites by means of bending tests on three points. The results showed a gradual increase in modulus with increasing percentage of PET in smaller particle sizes, resulting in significantly reducing deformation in relation to the matrix. [ABSTRACT FROM AUTHOR]

The Construction Industry as a generator of waste, have an important role in shaping the future, from the moment that embodies the culture of responsibility to preserve the environment. Even if properly manage the waste is still a problem faced by society in recent years to see changes in the construction industry, by introducing new technologies into their construction processes. Whether the methods and techniques of rationalization, sorting and recycling of waste through the recycling process and, when responsibility for the final destination of the waste generated at the construction site. This study aims to outline a model that sequencie the main activities to be adopted by public and private agencies, to enable them to manage their waste in accordance with specific legislation. [ABSTRACT FROM AUTHOR]

Cytidine deaminase (CDA) deficiency causes pyrimidine pool disequilibrium. We previously reported that the excess cellular dC and dCTP resulting from CDA deficiency jeopardizes genome stability, decreasing basal poly(ADP-ribose) polymerase 1 (PARP-1) activity and increasing ultrafine anaphase bridge (UFB) formation. Here, we investigated the mechanism underlying the decrease in PARP-1 activity in CDA-deficient cells. PARP-1 activity is dependent on intracellular NAD+ concentration. We therefore hypothesized that defects of the NAD+ salvage pathway might result in decreases in PARP-1 activity. We found that the inhibition or depletion of nicotinamide phosphoribosyltransferase (NAMPT), the rate-limiting enzyme in the NAD+ salvage biosynthesis pathway, mimicked CDA deficiency, resulting in a decrease in basal PARP-1 activity, regardless of NAD+ levels. Furthermore, the expression of exogenous wild-type NAMPT fully restored basal PARP-1 activity and prevented the increase in UFB frequency in CDA-deficient cells. No such effect was observed with the catalytic mutant. Our findings demonstrate that (1) the inhibition of NAMPT activity in CDA-proficient cells lowers basal PARP-1 activity, and (2) the expression of exogenous wild-type NAMPT, but not of the catalytic mutant, fully restores basal PARP-1 activity in CDA-deficient cells; these results strongly suggest that basal PARP-1 activity in CDA-deficient cells decreases due to a reduction of NAMPT activity. [ABSTRACT FROM AUTHOR]